1. Gukovskaya, AS, Gukovsky, I. Which way to die: the regulation of acinar cell death in pancreatitis by mitochondria, calcium, and reactive oxygen species. Gastroenterology 2011; 140(7): 1876–1880.
Google Scholar |
Crossref |
Medline2. Ji, L, Li, L, Qu, F, et al. Hydrogen sulphide exacerbates acute pancreatitis by over‐activating autophagy via AMPK/mTOR pathway. J Cell Mol Med 2016; 20(12): 2349–2361.
Google Scholar |
Crossref |
Medline3. Kang, R, Lotze, MT, Zeh, HJ, et al. Cell death and DAMPs in acute pancreatitis. Mol Med 2014; 20(1): 466–477.
Google Scholar |
Crossref |
Medline4. Norman, JG, Franz, MG, Fink, GS, et al. Decreased mortality of severe acute pancreatitis after proximal cytokine blockade. Ann Surg 1995; 221(6): 625–634.
Google Scholar |
Crossref |
Medline5. Hoque, R, Sohail, M, Malik, A, et al. TLR9 and the NLRP3 Inflammasome Link Acinar Cell Death With Inflammation in Acute Pancreatitis. Gastroenterology 2011; 141(1): 358–369.
Google Scholar |
Crossref |
Medline6. Labbé, K, Saleh, M. Cell death in the host response to infection. Cel Death Differ 2008; 15(9): 1339–1349.
Google Scholar |
Crossref |
Medline |
ISI7. Shi, J, Gao, W, Shao, F. Pyroptosis: Gasdermin-mediated programmed necrotic cell death. Trends Biochem Sci 2017; 42(4): 245–254.
Google Scholar |
Crossref |
Medline8. Feng, C, Su, X, Zhou, X, et al. Early peritoneal lavage with ulinastatin improves outcome and enhances multi-organ protection in a model of severe acute pancreatitis. Exp Therapeutic Medicine 2015; 9(4): 1171–1177.
Google Scholar |
Crossref |
Medline9. Liu, R, Qi, H, Wang, J, et al. Ulinastatin activates the renin-angiotensin system to ameliorate the pathophysiology of severe acute pancreatitis. J Gastroenterol Hepatol 2014; 29(6): 1328–1337.
Google Scholar |
Crossref |
Medline10. Schmidt, J, Rattner, DW, Lewandrowski, K, et al. A better model of acute pancreatitis for evaluating therapy. Ann Surg 1992; 215(1): 44–56.
Google Scholar |
Crossref |
Medline |
ISI11. Louhimo, JM, Steer, ML, Perides, G. Necroptosis Is an Important Severity Determinant and Potential Therapeutic Target in Experimental Severe Pancreatitis. Cell Mol Gastroenterol Hepatol 2016; 2(4): 519–535.
Google Scholar |
Crossref |
Medline12. Cookson, BT, Brennan, MA. Pro-inflammatory programmed cell death. Trends Microbiol 2001; 9(3): 113–114.
Google Scholar |
Crossref |
Medline |
ISI13. Shao, G, Zhou, Y, Song, Z, et al. The diffuse reduction in spleen density: an indicator of severe acute pancreatitis?. Biosci Reports 2017; 37(1): BSR20160418.
Google Scholar |
Crossref |
Medline14. Ferrero-Andrés, A, Panisello-Roselló, A, Roselló-Catafau, J, et al. NLRP3 inflammasome-mediated inflammation in acute pancreatitis. Int J Mol Sci 2020; 21(15): 5386.
Google Scholar |
Crossref15. Mazzolini, G, Sowa, J-P, Canbay, A. Cell death mechanisms in human chronic liver diseases: a far cry from clinical applicability. Clin Sci 2016; 130(23): 2121–2138.
Google Scholar |
Crossref16. Fink, SL, Cookson, BT. Caspase-1-dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages. Cell Microbiol 2006; 8(11): 1812–1825.
Google Scholar |
Crossref |
Medline17. Schroder, K, Tschopp, J. The inflammasomes. Cell 2010; 140(6): 821–832.
Google Scholar |
Crossref |
Medline |
ISI18. Yazdi, AS, Guarda, G, D’Ombrain, MC, et al. Inflammatory caspases in innate immunity and inflammation. J Innate Immun 2010; 2(3): 228–237.
Google Scholar |
Crossref |
Medline19. Brydges, SD, Broderick, L, McGeough, MD, et al. Divergence of IL-1, IL-18, and cell death in NLRP3 inflammasomopathies. J Clin Invest 2013; 123(11): 4695–4705.
Google Scholar |
Crossref |
Medline20. Strowig, T, Henao-Mejia, J, Elinav, E, et al. Inflammasomes in health and disease. Nature 2012; 481(7381): 278–286.
Google Scholar |
Crossref |
Medline |
ISI21. Shi, J, Zhao, Y, Wang, K, et al. Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death. Nature 2015; 526(7575): 660–665.
Google Scholar |
Crossref |
Medline22. Ding, J, Wang, K, Liu, W, et al. Pore-forming activity and structural autoinhibition of the gasdermin family. Nature 2016; 535(7610): 111–116.
Google Scholar |
Crossref |
Medline23. Shi, L, Ye, L, Liu, P, et al. Ulinastatin inhibits apoptosis induced by serum deprivation in mesenchymal stem cells. Mol Medicine Reports 2019; 19(3): 2397–2406.
Google Scholar |
Crossref |
Medline24. Hu, XH, Zhang, HY, Ge, YL, et al. [Protective effects of ulinastatin against multiple organic damage after severe burn injury: experimental and clinic studies]. Zhonghua Yi Xue Za Zhi 2005; 85(41): 2889–2894.
Google Scholar |
Medline25. Broz, P, Pelegrín, P, Shao, F. The gasdermins, a protein family executing cell death and inflammation. Nat Rev Immunol 2020; 20(3): 143–157.
Google Scholar |
Crossref |
Medline26. He, HW, Zhang, H. The efficacy of different doses of ulinastatin in the treatment of severe acute pancreatitis. Ann Palliat Med 2020; 9(3): 730–737.
Google Scholar |
Crossref |
Medline27. Pan, Y, Fang, H, Lu, F, et al. Ulinastatin ameliorates tissue damage of severe acute pancreatitis through modulating regulatory T cells. J Inflamm 2017; 14: 7.
Google Scholar |
Crossref
Comments (0)